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Pharmacology: The target site of aescin is the vascular wall. In the case of inflammatory raised permeability, escin inhibits exudation by reducing extravasation of fluid into the tissue spaces, and it accelerates reabsorption of the existing oedema. The mechanism of action is based on a change in the permeability of the capillary openings involved. Furthermore, escin promotes capillary resistance, inhibits inflammatory processes and improves microcirculation.
Diethylamine salicylate (DEAS) has a pronounced analgesic effect. It penetrates freely through the skin and exerts its analgesic action in the depths of the affected area. The additional antiphlogistic action of DEAS enhances the anti-inflammatory effect of aescin and thus combats the causal factors of the course of disease.
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